Moreover, a element of endothelial dysfunction has been proposed, which may contribute to irregular vessel reactivity. However, testosterone in physiologic doses could even be useful in sufferers with angina , have higher ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood circulate in men with established coronary artery illness . Therefore, an interaction of endogenous elements of the patient’s threat profile in addition to the testosterone dosing may be problems with whether adverse events occur.
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement therapy and proposals for monitoring. Testosterone replacement in hypogonadal men with angina improves ischaemic threshold and high quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.
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Erythrocytosis has been reported as a possible impact of testosterone, which may be beneficial in patients with anemia. The best danger of erythrocytosis occurs with injectable types of testosterone in contrast with transdermal preparations, especially in supraphysiologic doses. An increase of as much as 44% of the baseline hematocrit has been reported .
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We report a case of an acute ST-segment elevation myocardial infarction in a affected person with traditional cardiac risk factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient also had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent successful percutaneous intervention of the best coronary artery. Physicians and patients ought to pay consideration to the scientific consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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